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Actual results and the timing of events could differ materially from those anticipated in such forward-looking statements as a result of these risks and uncertainties, which include, without limitation the risk factors described in the Company's US Securities and Exchange Commission filings and reports Commission File No.
This presentation discusses product candidates that are under clinical study, and which have not yet been approved for marketing by the US Food and Drug Administration. No representation is made as to the safety or effectiveness of these product candidates for the use for which such product candidates are being studied. Caution should be exercised when comparing data across trials of different products and product candidates.
Differences existing between trial designs and patient populations and characteristics. Results Data presented in this poster confirm prior results of eblasakimab dampening neuronal sensitization caused by IL-4 and IL on human neurons. In addition, IL sensitized hDRG neurons to pro-adrenomedullin peptide PAMP20 , demonstrating that the itch-specific MRGPRX2 receptor is expressed and functional in human sensory neurons, suggesting an enhancer role of Type 2 cytokines in a wide variety of itch-associated diseases.
IL-4 induced spontaneous activity SA in hDRG neurons under inflammatory conditions, which was significantly reduced by eblasakimab. This may underly the mechanistic basis for altered neuronal sensitivity in inflammatory conditions. These data suggest that IL and IL-4 may play distinct roles in affecting neuronal activity and eblasakimab was shown to block the effects of both cytokines.
The posters presented at the conference are available to access here. References Silverberg JI, et al. Ann Allergy Asthma Immunol. Junttila IS. Devillers, A. Clinical and Experimental Dermatology, Jung, AR. Zheng H, et al. Sci Rep. Amarbayasgalan T, et al. Int Arch Allergy Immunol. Oetjen L, et al. Miron Y, et al. J Allergy Clin Immunol.
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